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In recent years, there has been a growing global interest in the idea of drug repurposing-the practice of using existing medications for new therapeutic purposes. Within this movement, a group of antiparasitic drugs, including Fenbendazole, Mebendazole, and Ivermectin, has captured attention for their potential relevance in cancer research. Originally developed to treat parasitic infections in animals and humans, these medications are now being explored in laboratory settings for their possible effects on cancer-related biological pathways. Much of the public awareness surrounding this topic can be traced to the widely shared story of Joe Tippens. His personal experience, combined with anecdotal reports from others, has sparked curiosity, discussion, and debate across patient communities, researchers, and healthcare professionals. While these stories have inspired hope, they have also highlighted the need for careful examination of the science behind such claims. This guide is designed to provide a structured, research-focused overview of antiparasitic protocols as they relate to cancer. It does not promote or prescribe treatment but instead aims to help readers understand the scientific theories, biological mechanisms, and current evidence associated with these drugs. By exploring both the potential and the limitations, this book encourages a balanced and informed perspective. The discussion begins with the origins of the protocol and the emergence of online communities that contributed to its spread. It then examines the parasite-cancer hypothesis, a controversial idea suggesting that certain infections or parasite-like mechanisms may play a role in cancer development. While not widely accepted in mainstream oncology, this hypothesis has influenced how some individuals interpret the potential role of antiparasitic drugs. From there, the guide explores the broader field of drug repurposing in oncology, highlighting why existing medications are being reconsidered as possible cancer therapies. The pharmacological profiles of fenbendazole, mebendazole, and ivermectin are examined in detail, including their known mechanisms of action and the ways they may interact with cancer-related processes such as cell division, metabolism, and immune response.